5B90

Certain Specified Nutrient Excesses ICD 11 Codes

Definition of Certain Specified Nutrient Excesses: Any disease caused by an excess of specific nutrients. Confirmation is by blood test.

Vitamin excesses

ICD 11 Code For Vitamin excesses

  5B90  Vitamin excesses

5B90.0 Hypervitaminosis A

Definition of Hypervitaminosis A: Because vitamin A is fat soluble and can be stored, primarily in the liver, routine consumption of large amounts of vitamin A over a period of time can result in toxic symptoms, including liver damage, bone abnormalities and joint pain, alopecia, headaches, vomiting, and skin desquamation. Hypervitaminosis A appears to be due to abnormal transport and distribution of vitamin A and retinoids caused by overloading of the plasma transport mechanisms. Very high single doses can cause transient acute toxic symptoms that may include bulging fontanelles in infants; headaches in older children and adults; and vomiting, diarrhoea, loss of appetite, and irritability in all age groups. Rarely does toxicity occur from ingestion of food sources of preformed vitamin A. When this occurs, it usually results from very frequent consumption of liver products.

Coded Elsewhere:

  • Pseudotumour Cerebri related to Hypervitaminosis A (8D41.2)

5B90.1 Hypercarotenaemia

Definition of Hypercarotenaemia: Because vitamin A is fat soluble and can Excessive intake of carotenoids is not associated with toxicity but can cause yellow coloration of the skin that disappears when intake is reduced. This disorder is especially likely to occur in children with liver disease, diabetes mellitus or hypothyroidism, and in those who do not have enzymes that metabolize carotenoids.

5B90.2 Hypervitaminosis D

Definition of Hypervitaminosis D: Hypervitaminosis D is secondary to excessive intake of vitamin D. It can occur with long-term high intake or with a substantial, acute ingestion. Excess amounts result in abnormally high concentrations of calcium and phosphate in the serum. The signs and symptoms of vitamin D intoxication are secondary to hypercalcaemia. Gastrointestinal manifestations include nausea, vomiting, constipation, abdominal pain and pancreatitis. Possible cardiac findings are hypertension, decreased Q-T interval and arrhythmias. The central nervous system effects of hypercalcaemia include lethargy, hypotonia, confusion, disorientation, depression, psychosis, hallucinations and coma. Hypercalcaemia impairs renal concentrating mechanisms, which can lead to polyuria, dehydration and hypernatremia. Hypercalcaemia can also lead to acute renal failure, nephrolithiasis and nephrocalcinosis, which can result in chronic renal insufficiency. Deaths are usually associated with arrhythmias or dehydration.

5B90.3 Megavitamin-B6 syndrome

Definition of Megavitamin-B6 syndrome: A disease caused by an excess of vitamin B6. This disease is characterised by progressive sensory ataxia, diminished or absent tendon reflexes, and impaired sense of touch, temperature and pain. Confirmation is by blood test.

Coded Elsewhere:

  • Peripheral neuropathy due to vitamin B6 hyperalimentation (8D41.0)

5B90.Y Other specified vitamin excess

5B90.Z Unspecified vitamin excesses

Mineral excesses

ICD 11 Code For Mineral excesses

  5B91  Mineral excesses

Coded Elsewhere:

5B91.0 Hypercalcaemia

Definition of Hypercalcaemia: Hypercalcaemia is a condition caused by increased calcium levels. The higher the calcium levels and the faster its level rises, the more severe will be the symptoms. When present, symptoms are caused by dehydration secondary to urinary losses of calcium, water and other electrolytes, and to an increase in membrane potential caused by the elevation in extracellular fluid ionized calcium concentration. Patients with moderate to severe hypercalcaemia often complain of nausea and vomiting, symptoms likely related to dehydration as well as to the effects of the hypercalcaemia on central nervous system function. Because hypercalcaemia tends to hyperpolarize membranes, a range of neurologic and neuromuscular signs and symptoms can occur. Patients with mild hypercalcaemia often complain of fatigue, depressed mood and asthenia. Gastrointestinal motility is impaired; this commonly results in constipation.

Coded Elsewhere:

  • Myopathy due to hypercalcaemia (8D41.1)

5B91.1 Zinc excess

Definition of Zinc excess: Adverse effects associated with chronic intake of supplemental zinc include suppression of immune response, decrease in high-density lipoprotein (HDL) cholesterol and reduced copper status. Acute adverse effects of excess zinc include epigastric pain, nausea, vomiting, loss of appetite, abdominal cramps, diarrhoea, headaches and gastrointestinal distress.

Coded Elsewhere:

  • Myelopathy due to excess of zinc (8D41.Y)

5B91.2 Sodium chloride excess

Definition of Sodium chloride excess: The main adverse effect of increased sodium chloride in the diet is increased blood pressure, which is a major risk factor for cardiovascular-renal diseases. However, evidence from a variety of studies, including observational studies and clinical trials, has demonstrated heterogeneity in the blood pressure responses to sodium intake. Those individuals with the greatest reductions in blood pressure in response to decreased sodium intake are termed “salt sensitive”.

5B91.3 Fluorine excess

Definition of Fluorine excess: The primary adverse effects associated with chronic, excess fluoride intake are enamel and skeletal fluorosis. Enamel fluorosis is a dose-response effect caused by fluoride intake during the pre-eruptive development of teeth. The development of skeletal fluorosis and its severity is directly related to the level and duration of exposure. The clinical signs in advanced stages may include dose-related calcification of ligaments, osteosclerosis, exostoses, possibly osteoporosis of long bones, muscle wasting and neurological defects due to hypercalcification of vertebrae.

Coded Elsewhere:

  • Dental enamel fluorosis (DA07.0)

5B91.4 Aluminium excess

Definition of Aluminium excess: Patients receiving long-term parenteral nutrition are at increased risk of aluminium toxicity because of bypass of the gastrointestinal tract during parenteral nutrition infusion. Complications of aluminium toxicity include metabolic bone disease, aluminium-associated encephalopathy in adults and impaired neurological development in preterm infants.

5B91.5 Manganese excess

Definition of Manganese excess: Manganese toxicity in humans is a well-recognised occupational hazard for people who inhale manganese dust. High concentrations of circulating manganese as a result of total parenteral nutrition have also been associated with manganese toxicity. People with chronic liver disease have neurological pathology and behavioural signs of manganese neurotoxicity, probably because elimination of manganese in bile is impaired. The most prominent effect is central nervous system pathology, especially in the extra-pyramidal motor system. The lesions and symptoms are similar to those of Parkinson’s disease.

Coded Elsewhere:

  • Dementia or parkinsonism due to manganese toxicity (6D84.Y)

5B91.Y Other specified mineral excess

5B91.Z Unspecified mineral excess

Other specified nutrient excesses

ICD 11 Code For Other specified nutrient excesses

  5B9Y  Other specified nutrient excesses

Certain specified nutrient excesses unspecified

ICD 11 Code For Certain specified nutrient excesses unspecified

  5B9Z  Certain specified nutrient excesses unspecified

Other specified overweight obesity or specific nutrient excesses

ICD 11 Code For Other specified overweight obesity or specific nutrient excesses

  5C1Y  Other specified overweight obesity or specific nutrient excesses

Overweight obesity or specific nutrient excesses unspecified

ICD 11 Code For Overweight obesity or specific nutrient excesses unspecified

  5C1Z  Overweight obesity or specific nutrient excesses unspecified

Other specified nutritional disorders

ICD 11 Code For Other specified nutritional disorders

  5C3Y  Other specified nutritional disorders

Nutritional disorders unspecified

ICD 11 Code For Nutritional disorders unspecified

  5C3Z  Nutritional disorders unspecified

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